NESA Biology Epidemiology
5 sample questions with marking guides and sample answers · Avg. score: 100%
Over 12 months, the prevalence of a non-infectious disease will increase in a population if
the total population increases.
disease recovery time decreases.
the incidence rate of the disease decreases.
the survival time of individuals with the disease increases.
Parkinson's disease can potentially be treated using cell replacement therapy. The purpose of using this therapy would be to replace the cells that
would normally function within the cerebral cortex.
would normally function within the cerebellum.
produce dopamine in the brain.
produce noradrenaline in the brain.
Christian, a 40-year-old man, was recently diagnosed with Type 2 diabetes. Christian was classified as obese according to his body mass index and his fasting blood glucose level was very high being measured at 10.3 mmol L. In addition to changes to his diet and increased exercise, Christian's doctor prescribed a drug to promote weight loss and lower Christian's blood glucose levels. This drug mimics a natural water-soluble hormone in the human body, enhancing the action of insulin and reducing glucagon levels.
Explain how the following glands maintain blood glucose levels during fasting: ...
Outline how water-soluble hormones influence their target cells.
... pancreas
... adrenal glands
Explain why a patient with Type 1 diabetes would not be prescribed the drug from the above scenario to manage their blood glucose levels.
Changes to the amount of thyroxine being released from the thyroid gland can have major impacts on the functioning of the body.
Name each of the disorders that may lead to an over-secretion or under-secretion of thyroxine; describe how the over-secretion or under-secretion impacts on the body; and explain how each disorder can be treated.
Explain the role of the liver in the maintenance of blood glucose levels.
A promising experimental therapy being used to treat Parkinson's disease involves the use of
stem cells to replace damaged or dysfunctional dopamine-producing neurons.
gene therapy to fix mutated DNA sequences in brain cells.
protein blockers to prevent the building up of amyloid plaques in the brain.
invasive brain stimulation therapy to activate dysfunctional neurons.